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Ellen Foxman

@ellenfoxman.bsky.social
2.6K followers 1.1K following 47 posts

Associate Professor, Immunologist, Physician-Scientist. Studying human immunology, innate immunity, host-virus interactions for better diagnosis, treatment, prevention. Opinions are my own.

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Reposted by Ellen Foxman
Waggoner Lab @labwaggoner.bsky.social · 04/03/2026
Rhinovirus triggers distinct host responses through differential engagement of epithelial innate immune signaling @ellenfoxman.bsky.social @yalepress.bsky.social www.cell.com/cell-press-b... @cellpressblue.bsky.social
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Reposted by Ellen Foxman
Thelonevirologist @thelonevirologist.bsky.social · 02/02/2026
Mechanistic insights into the impact of prenatal viral infections on maternal and offspring immunity www.nature.com/articles/s44...
nature.com
Mechanistic insights into the impact of prenatal viral infections on maternal and offspring immunity - npj Viruses
npj Viruses - Mechanistic insights into the impact of prenatal viral infections on maternal and offspring immunity
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Reposted by Ellen Foxman
PLOS Biology @plosbiology.org · 28/01/2026
#Interferons protect against #ViralInfection, but can hinder tissue repair - how? @ellenfoxman.bsky.social &co show that high IFN levels suppress #BronchialCell migration & proliferation during tissue repair via JAK-driven LATS1 activation, independent of STAT1 @plosbiology.org 🧪 plos.io/4k1pyKA
Top: Type III interferon alters the nuclear localization of the transcriptional co-regulator YAP during wound healing of human bronchial epithelial cells.  Micrographs show localization or YAP (red) relative to the cell nucleus (blue) in cells adjacent to a scratch in the monolayer (dark area, right of images).  In untreated cultures, YAP is present in most nuclei (purple nuclei, left panel) whereas in the presence of Type III interferon, the percentage of cells with nuclear YAP is greatly reduced (blue nuclei, right panel).  Image credit: Krupakar Subramaniam. Bottom: Model. Type III and Type I IFN receptor signaling activate LATS1 to curtail tissue repair during acute viral infection. Left-hand panel:  During acute viral infection, high concentrations of Type III and/or Type I IFN receptor signaling trigger JAK-dependent, STAT1-independent phosphorylation of LATS1, leading to phosphorylation and degradation of YAP and reduced tissue repair activities mediated by YAP target genes. In rapidly proliferating cell types, active LATS1/2 also blocks repair in part via a p53-dependent mechanism. Right-hand panel: During resolution of viral infection, lower concentrations of Type III and/or Type I IFN may continue to activate STAT1-mediated antiviral defenses but no longer activate LATS1/2, allowing epithelial repair to resume.
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Ellen Foxman @ellenfoxman.bsky.social · 02/02/2026
Thank you to Bob McDonald for hosting me on @CBCRadioCanada to discuss our work @yaleschoolofmed.bsky.social on what leads to variation in common cold virus infections and how we tackled this using nasal organoids. Listen here: www.cbc.ca/listen/live-...
cbc.ca
© CBC/Radio-Canada 2026. All rights reserved.
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Ellen Foxman @ellenfoxman.bsky.social · 31/01/2026
Excited to see this beautiful image of nasal #organoids featured as biomedical Image of the day by @cellpress.bsky.social 🙏🙏🙏 See full paper @cellpressblue.bsky.social #respiratory #lungs #EpithelialMechanics #IdSky #MedSky #ImmunoSky
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Ellen Foxman @ellenfoxman.bsky.social · 29/01/2026
#Epithelialmechanics fans...interferons protect against viruses but also impair recovery post-infection. In new work @plosbiology.org we show that interferons block epithelial repair by co-opting the Hippo pathway #innateimmunity #lungs #respiratory journals.plos.org/plosbiology/...
journals.plos.org
Dose-dependent activation of the Hippo pathway by Type I and Type III interferons suppresses tissue repair by human bronchial epithelial cells
Interferons (IFNs) initiate antiviral defence when infected cells sense a virus protecting against infection via JAK/STAT signaling, but can hinder recovery by impairing tissue repair by an unknown me...
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Ellen Foxman @ellenfoxman.bsky.social · 29/01/2026
www.thedoctorwillseeyounow.com/content/infe... #lungs #MedSky #IDsky #ImmunoSky
thedoctorwillseeyounow.com
How Your Body Fights Off the Cold Virus
Ever wonder why some people get really bad colds and others never get sick? Modeling the cells in human noses and lungs suggests an answer.
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Reposted by Ellen Foxman
Thelonevirologist @thelonevirologist.bsky.social · 28/01/2026
Frequent presymptomatic household transmission of influenza A but not influenza B virus www.nature.com/articles/s44...
nature.com
Frequent presymptomatic household transmission of influenza A but not influenza B virus - Nature Health
Based on two waves of data collection from 748 households in Hong Kong, this analysis sheds light on the number of transmission events that occurred before manifestation of symptoms in influenza A and...
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Reposted by Ellen Foxman
PLOS Biology @plosbiology.org · 27/01/2026
#Interferons protect against #ViralInfection, but can hinder tissue repair - how? @ellenfoxman.bsky.social &co show that high IFN levels suppress #BronchialCell migration & proliferation during tissue repair via JAK-driven LATS1 activation, independent of STAT1 @plosbiology.org 🧪 plos.io/4k1pyKA
Top: Type III interferon alters the nuclear localization of the transcriptional co-regulator YAP during wound healing of human bronchial epithelial cells.  Micrographs show localization or YAP (red) relative to the cell nucleus (blue) in cells adjacent to a scratch in the monolayer (dark area, right of images).  In untreated cultures, YAP is present in most nuclei (purple nuclei, left panel) whereas in the presence of Type III interferon, the percentage of cells with nuclear YAP is greatly reduced (blue nuclei, right panel).  Image credit: Krupakar Subramaniam. Bottom: Model. Type III and Type I IFN receptor signaling activate LATS1 to curtail tissue repair during acute viral infection. Left-hand panel:  During acute viral infection, high concentrations of Type III and/or Type I IFN receptor signaling trigger JAK-dependent, STAT1-independent phosphorylation of LATS1, leading to phosphorylation and degradation of YAP and reduced tissue repair activities mediated by YAP target genes. In rapidly proliferating cell types, active LATS1/2 also blocks repair in part via a p53-dependent mechanism. Right-hand panel: During resolution of viral infection, lower concentrations of Type III and/or Type I IFN may continue to activate STAT1-mediated antiviral defenses but no longer activate LATS1/2, allowing epithelial repair to resume.
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Reposted by Ellen Foxman
Ivan Zanoni @lozanzi.bsky.social · 27/01/2026
#InterferonFriendsOrFoes? Another cool one @ellenfoxman.bsky.social &friends out @plosbiology.org about how excessive #IFNs delay repair by activating LATS & degrading YAP/TAZ in the Hippo 🦛 pathway! journals.plos.org/plosbiology/... 👇👇👇
journals.plos.org
Dose-dependent activation of the Hippo pathway by Type I and Type III interferons suppresses tissue repair by human bronchial epithelial cells
Interferons (IFNs) initiate antiviral defence when infected cells sense a virus protecting against infection via JAK/STAT signaling, but can hinder recovery by impairing tissue repair by an unknown me...
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Reposted by Ellen Foxman
PLOS Biology @plosbiology.org · 27/01/2026
#Interferons protect against #ViralInfection, but can hinder tissue repair - how? @ellenfoxman.bsky.social &co show that high IFN levels suppress #BronchialCell migration & proliferation during tissue repair via JAK-driven LATS1 activation, independent of STAT1 @plosbiology.org 🧪 plos.io/4k1pyKA
Top: Type III interferon alters the nuclear localization of the transcriptional co-regulator YAP during wound healing of human bronchial epithelial cells.  Micrographs show localization or YAP (red) relative to the cell nucleus (blue) in cells adjacent to a scratch in the monolayer (dark area, right of images).  In untreated cultures, YAP is present in most nuclei (purple nuclei, left panel) whereas in the presence of Type III interferon, the percentage of cells with nuclear YAP is greatly reduced (blue nuclei, right panel).  Image credit: Krupakar Subramaniam. Bottom: Model. Type III and Type I IFN receptor signaling activate LATS1 to curtail tissue repair during acute viral infection. Left-hand panel:  During acute viral infection, high concentrations of Type III and/or Type I IFN receptor signaling trigger JAK-dependent, STAT1-independent phosphorylation of LATS1, leading to phosphorylation and degradation of YAP and reduced tissue repair activities mediated by YAP target genes. In rapidly proliferating cell types, active LATS1/2 also blocks repair in part via a p53-dependent mechanism. Right-hand panel: During resolution of viral infection, lower concentrations of Type III and/or Type I IFN may continue to activate STAT1-mediated antiviral defenses but no longer activate LATS1/2, allowing epithelial repair to resume.
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Ellen Foxman @ellenfoxman.bsky.social · 24/01/2026
Interested in the common cold virus? Check out this clip from the Naked Scientist podcast about our recent study in which we observed thousands of nasal cells interact to cause (or stop) symptoms of common cold infections. #MedSky #IDsky #lungs www.thenakedscientists.com/articles/int...
thenakedscientists.com
Nose-in-a-dish: Scientists recreate common cold in culture
Rhinoviruses really nose how to change cells...
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Reposted by Ellen Foxman
Ivan Zanoni @lozanzi.bsky.social · 22/01/2026
Fantastic work @ellenfoxman.bsky.social &co! If you block the #InterferonPower, Rhinovirus activates NFkB & NLRP1 #inflammasome leading to IL1b secretion & excessive mucus production!
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Ellen Foxman @ellenfoxman.bsky.social · 20/01/2026
Rhinoviruses cause common cold & asthma attacks but are also often benign. Using nasal organoids, we learned how the community of cells in the lining the nasal passages coordinates to respond to rhinovirus and which responses lead to excess mucus & inflammation. www.cell.com/cell-press-b...
cell.com
Rhinovirus triggers distinct host responses through differential engagement of epithelial innate immune signaling
Rhinoviruses are the most frequent cause of common colds and also a major cause of respiratory distress in high-risk groups. Using single-cell sequencing of rhinovirus-infected nasal epithelial organo...
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Reposted by Ellen Foxman
Cell Press @cellpress.bsky.social · 19/01/2026
Cells in our nasal passages can detect common cold viruses and mount a defense before we catch it. spkl.io/63321Ao5fV @ellenfoxman.bsky.social & colleagues @cellpressblue.bsky.social
Rhinoviruses are the most frequent cause of common colds but also a major cause of respiratory distress in high-risk groups. Using single-cell sequen cing of rhinovirus-i nfected nasal epithelial organoids, Foxman and colleagues elucidate effective and maladaptive innate immune responses to rhinovirus infection and define targets for intervention
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Nick Hopkinson @copddoc.bsky.social · 02/01/2026
Happy Birthday Christy Turlington.
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Victor Tatarskiy @greenrat.bsky.social · 02/10/2025
Neat trick if you polycolonal ab's suck. Incubate them with fixed cells with a KO of your protein of interest, then spin. Protocol here: www.med.upenn.edu/markslab/ass... I was amazed how well it worked on first try (I'm sure that I can completely eliminate unspecific bands) #WesternBlot #cellsky
On the left - western blot of B16F10 cells wt and KO for CDK8. Our in house produced antibodies give a lot of unspecific bands. On the right same probes with antibodies preincubated with fixed CDK8 KO cells - there is a specific band and faint unspecific bands, which can be probably eliminated with increase of amount of KO cells.
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Reposted by Ellen Foxman
Marc Veldhoen @marcveld.bsky.social · 09/10/2025
A cGAS-mediated mechanism in naked mole-rats potentiates DNA repair and delays aging Naked mole rats get much older than closely related mamals of a similar size. cGAS mutations may have an important role in this. www.science.org/doi/... 1/9
science.org
A cGAS-mediated mechanism in naked mole-rats potentiates DNA repair and delays aging
Efficient DNA repair might make possible the longevity of naked mole-rats. However, whether they have distinctive mechanisms to optimize functions of DNA repair suppressors is unclear. We find that naked mole-rat cyclic guanosine monophosphate–adenosine ...
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Ellen Foxman @ellenfoxman.bsky.social · 13/10/2025
Happy to share this review just out in Current Opinion in Immunology on innate immunity in the respiratory tract - also check out the other reviews in this collection on Intrinsic Immunity edited by John MacMicking #ImmunoSky #IDSky #pulmsky #MedSky www.sciencedirect.com/science/arti...
sciencedirect.com
Layers of defense: protection from respiratory viruses by epithelial-intrinsic immunity
A central challenge in defending mucosal barriers is protecting against pathogens while also limiting excessive inflammation. Respiratory viruses are …
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Ellen Foxman @ellenfoxman.bsky.social · 23/06/2025
#COVID19 revealed the urgent need for better ways to screen for & rule out respiratory virus infection. We present a nasal biomarker screening strategy to prevent & manage outbreaks @eBiomedicine.bsky.social #IDSky #MedSky #lungs @yaleschoolofmed.bsky.social www.thelancet.com/journals/ebi...
thelancet.com
Nasal biomarker testing to rule out viral respiratory infection and triage samples: a test performance study
These results demonstrate the utility of a nasopharyngeal biomarker to rule out respiratory infection, with potential applications in outbreak management and/or routine screening in high-risk settings...
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Reposted by Ellen Foxman
Jeremy Berg @jeremymberg.bsky.social · 31/05/2025
ASSIGNMENT: COMMENT ON SCHEDULE F 1 week left to comment about Schedule F, potentially making NIH institute and center director essentially political appointees. www.regulations.gov/commenton/OP... 1/n
regulations.gov
Regulations.gov
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Hensley Lab @scottehensley.bsky.social · 30/05/2025
Nice coverage of our H5 mRNA work by Jon LaPook tonight on @cbseveningnews.bsky.social www.cbsnews.com/video/the-sc...
cbsnews.com
The scientists aiming to stay ahead of bird flu on dairy farms and prevent a human pandemic
So far, the only people to come down with bird flu got it from animals. Dr. Jon LaPook reports on how research is underway to keep bird flu from mutating into a form that could spread from person to p...
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Reposted by Ellen Foxman
David Shiffman, Ph.D. 🦈 @whysharksmatter.bsky.social · 28/05/2025
Are you a scientist and not sure what to do to help with all the awfulness all around us? Inspired by Vietnam era activism, @standupforscience.bsky.social is hosting a series of "teach ins," in which scientists share a bit about their area of expertise and why it's important with the public. 🧪🦑🌎
Flier for my talk Friday 5/30 at 7 p.m.
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Hensley Lab @scottehensley.bsky.social · 21/05/2025
Check out this news story that @nature.com wrote describing our recent preprint where we tested our H5 mRNA vaccine in calves. The data look good and we are currently testing the vaccine in lactating cattle...more to come!
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Caroleeena @caroleeena.bsky.social · 19/05/2025
The Nile from the International Space Station:
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Jonathan Slater @jonathanslater.bsky.social · 19/05/2025
Once in a while. Good morning Blue Sky
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Dr. Lucky Tran @luckytran.com · 05/04/2025
Spectacular view of 100,000 people attending the #HandsOff march in NYC
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Dr. Sunny Shin @sunnyshinlab.bsky.social · 02/04/2025
Thrilled to share our @elife.bsky.social paper "Inflammasomes primarily restrict cytosolic Salmonella replication within human macrophages" by 1st author Dr. @marisaegan.bsky.social (@cambupenn.bsky.social alum, now faculty @swarthmorecollege.bsky.social)! elifesciences.org/articles/90107 🧵(1/n)
elifesciences.org
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Carl Zimmer @carlzimmer.com · 29/03/2025
Top F.D.A. Vaccine Official Resigns, Citing Kennedy’s ‘Misinformation and Lies’ Gift link: nyti.ms/3DTuvVr
nyti.ms
Top FDA Vaccine Official Resigns, Citing Kennedy’s ‘Misinformation and Lies’ (Gift Article)
Dr. Peter Marks, a veteran of the agency, wrote that undermining confidence in vaccines is irresponsible and a danger to public health.
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Carolyn Bertozzi @carolynbertozzi.bskyverified.social · 27/03/2025
We are seeking to hire a lab tech, this is an HHMI funded position, please share the job posting: hhmi.wd1.myworkdayjobs.com/en-US/Extern...
hhmi.wd1.myworkdayjobs.com
Lab Administration Research Technician- Bertozzi Lab
Primary Work Address: Beckman Center, Stanford, CA, 94305-5323 Current HHMI Employees, click here to apply via your Workday account. We are currently looking for a highly motivated Lab Administration ...
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Nikolai Slavov @slavov-n.bsky.social · 22/03/2025
The difference between Accuracy & Precision.
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Maureen Barr @barrlab.bsky.social · 20/03/2025
In a science round table discussion yesterday, my NJ senator @kim.senate.gov suggested a strong argument for NIH and federal funding is global competitiveness. We US-based scientists should start weaving this into our narratives.
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Thiago Carvalho @cyrilpedia.bsky.social · 20/03/2025
'Neutrophils expressed notably low levels of STING, whereas forced expression led to uncontrolled systemic inflammation. During development in the thymus, Tcells dynamically & epigenetically restricted STING expression through DNMT1–mediated transcriptional silencing' #Immunology
science.org
Dynamic STING repression orchestrates immune cell development and function
Repression of STING expression in neutrophils and T lymphocytes is required for immune homeostasis and tumor response.
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Waggoner Lab @labwaggoner.bsky.social · 19/03/2025
@cp-trendsimmuno.bsky.social Using structure-function information from IFN-γ-binding proteins and biased agonists to uncouple immunostimulatory and immunosuppressive activities www.cell.com/trends/immun...
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Sam Wang @samwang.bsky.social · 19/03/2025
Today I'm filled with admiration for my university's president, Chris Eisgruber. He's standing up for Columbia and for universities as a home for freedom of ideas, debate, and thought - free of government attack. www.theatlantic.com/ideas/archiv...
theatlantic.com
The Cost of the Government’s Attack on Columbia
American universities have given the country prosperity and security. The Trump administration’s attack on academic freedom endangers all of that.
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Kalev Freeman @uvmtraumalab.bsky.social · 19/03/2025
My privilege to team up with FASEB President-elect Eric Kelley today. We visited the W. Virginia and #Vermont offices together to explain how science research saves lives, creates jobs and keeps the USA competitive. #FASEBonTheHill #FundScience #NIH #MedSky #AcademicSky #UVMLarnerMed🧪
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Andrei Goga @andreigoga.bsky.social · 20/03/2025
UCSF, like all UC campuses, announced an immediate hiring freeze today. UCSF is the second largest employer in the city. There will be wide ripple effects in the Bay Area and beyond, including the future of biotech and pharma in the area. 🧪
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Nicole Bedera @nbedera.bsky.social · 18/03/2025
If there's one thing that domestic violence research has taught me, it's that abusers set up no-win scenarios. They punish you whether you cooperate with them or whether you push back. The only way out is to strengthen ties with people outside of the relationship who can create new, better options.
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Harmit Singh Malik @harmitmalik.bsky.social · 19/03/2025
Please consider nominating deserving colleagues (including yourselves) who would be excellent candidates for five open positions on the Council, including Nominations for President-elect, Secretary, Treasurer, and 2 Councilors. Please share widely! @official-smbe.bsky.social
Dear SMBE Members,

I am writing to solicit nominations for SMBE President-Elect, Secretary, Treasurer and 2 Councilors whose terms will begin on January 1, 2026. Each of these positions is for a duration of three years.

Council members play important roles in guiding the development of the society, soliciting and implementing programs that support our members, while enriching opportunities for young scientists from around the world. They also have a role in overseeing our two excellent journals (MBE and GBE) as well as our annual meeting, regional meetings, and satellite meetings.

Nominations will be reviewed by the nominations committee who will then put forward a slate of two candidates for each position for vote by the SMBE membership. Past and current council members are listed HERE.

Nominations (self-nominations are welcome) should consist of a brief statement in support of your suggestion (why this nomination, what the nominee can bring to SMBE, work subject, other biographical information). For Presidential nominees please include previous involvement with SMBE.

We ask the nominators to confirm explicitly that the person they are nominating has already confirmed their willingness to run for office.

Please send your nominations to Harmit Malik <hsmalik@fredhutch.org> AND to Emmanuelle Lerat <secretary.smbe@gmail.com> by April 18, 2025.

We look forward to hearing from you!

Sincerely yours,
Emmanuelle Lerat
Secretary of SMBE, on behalf of the Nominations Committee*

*The Nominations Committee is composed as follows:
Harmit S Malik (Chair) USA.
Xuming Zhou, China.
Mary O'Connell, UK.
Nicolas Galtier, France.
Deepa Agashe, India.
Emmanuelle Lerat (ex officio), France.
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Vaughn Smider, MD, PhD @vvsmider.bsky.social · 17/03/2025
I’m a biomedical researcher who lost his wife to cancer. Finding cures is personal to me. What’s happening at the NIH is devastating. Here’s my editorial in my hometown newspaper. www.vindy.com/opinion/edit...
vindy.com
DOGE ends America’s golden age of biomedical research
Biomedical research impacts all of us. I became curious about how drugs work after my mom was diagnosed with rheumatoid arthritis when I was a small boy. She received “gold shots” as treatment, which ...
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Carolyn Bertozzi @carolynbertozzi.bskyverified.social · 18/03/2025
Thank you @vvsmider.bsky.social for sharing this story.
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Ivan Zanoni @lozanzi.bsky.social · 16/03/2025
#WeekendRead! #InflammasomePower! @rauchlab.bsky.social &co show @jem.org that small intestinal tufts cells activate the NLRC4 inflammasome leading to PGD2-dependent IL-22 production by ILC3, protecting against Salmonella! rupress.org/jem/article/...
rupress.org
Enteric tuft cell inflammasome activation drives NKp46+ILC3 IL22 via PGD2 and inhibits Salmonella
PGD2 released after NAIP-NLRC4 inflammasome activation in tuft cells signals onto ILC3s and mediates host defense mechanisms against Salmonella Typhimurium
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Katri Bertram @katribertram.bsky.social · 17/03/2025
This 😭 via @economist.com
Photo from the Economist with quote “Public health is the mom of science - She does everything and no one noticea until she stops doing it”
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Gabriele Pollara @gpollara.bsky.social · 17/03/2025
Looking for a research assistant post in London? 🤔 Interested in how anti-cytokine auto-antibodies are associated with immune dysfunction and disease during ageing? 🎯 Then do consider applying for this exciting post 👇 #ImmunoSky #IDSky @uclmedsci.bsky.social www.ucl.ac.uk/work-at-ucl/...
ucl.ac.uk
UCL – University College London
UCL is consistently ranked as one of the top ten universities in the world (QS World University Rankings 2010-2022) and is No.2 in the UK for research power (Research Excellence Framework 2021).
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Needhi Bhalla 💅🏽 @needhibhalla.bsky.social · 15/03/2025
as I tell my Genetics class every year, this breakthrough is based on 70 years of NIH-funded, basic research, commercialized by biotech bsky.app/profile/need...
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Dave Baltrus 🦦 @surtlab.bsky.social · 15/03/2025
In my 25 years since high school I’ve seen 1. HIV/AIDS go from death sentence to manageable with drugs 2. Cystic fibrosis go from pretty much an extended death sentence to manageable with drugs 3. Sickle cell go from a horrible disease to (almost maybe) cured by gene therapy Simply incredible
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Hensley Lab @scottehensley.bsky.social · 13/03/2025
Today @natmedicine.bsky.social published our study showing the older individuals born prior to 1968 have more antibodies that cross-react to #H5N1 relative to younger individuals. If H5N1 causes a pandemic, children will likely be the most susceptible. 1/2 www.nature.com/articles/s41...
nature.com
Immune history shapes human antibody responses to H5N1 influenza viruses - Nature Medicine
H5N1 strain-specific antibodies are higher in older individuals and correlate more with birth year than with age, suggesting that younger individuals are potentially more likely to benefit from H5N1 v...
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Carl Zimmer @carlzimmer.com · 12/03/2025
West Texas measles outbreak spreads into Oklahoma www.nbcconnecticut.com/news/nationa...
nbcconnecticut.com
West Texas measles outbreak spreads into Oklahoma
A third state is now part of the growing measles outbreak in the U.S.
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Carl Zimmer @carlzimmer.com · 13/03/2025
White House Withdraws Nominee for C.D.C. Director Story by: @apoorvanyt.bsky.social and @sherylnyt.bsky.social Gift link: nyti.ms/3XQenKZ
nyti.ms
White House Withdraws Nominee for C.D.C. Director (Gift Article)
Dr. Dave Weldon was to have appeared on Thursday in a confirmation hearing before the Senate health committee. He has close ties to Robert F. Kennedy Jr., the new health secretary.
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Casanova Lab @casanovalab.bsky.social · 24/01/2025
1/ 📢 Register for the 2025 Henry Kunkel Society Meeting on "Genes & Immunity" and join us in Belgrade from April 10-12, 2025. www.henrykunkelsociety.org/meetings/ #HKS2025 #Immunology 🧬🔬
henrykunkelsociety.org
Meetings – Henry Kunkel Society
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