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Xinbo Wang

@xinbowang.bsky.social
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Reposted by Xinbo Wang
Yale Neuroscience @yaleneuro.bsky.social · 15/05/2026
"I always wanted to be a scientist." Pietro De Camilli’s journey from curious observation of nature as a child to understanding the molecular machinery of the brain has consistently been driven by an unwavering commitment to understanding life. @pdc-lab.bsky.social
medicine.yale.edu
Tending The Frontier: Pietro De Camilli and the Cell Biology of Neurons
Pietro De Camilli always wanted to be a scientist. He has spent his career bridging cell biology and neuroscience.
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Reposted by Xinbo Wang
Andrés Guillén Samander @aguillensamander.bsky.social · 31/03/2026
Our article is now out in its final form in @natcomms.nature.com. Thankful to reviewers for making our preprint better and of course all contributors and funders. Hopefully my first of many stories understanding the complex membrane cell biology of malaria parasites 🙌 www.nature.com/articles/s41...
nature.com
A bridge-like lipid transfer protein is critical for generation of invasive stages in malaria parasites - Nature Communications
Organelle biogenesis in the rapidly growing malaria parasite requires extensive membrane genesis and remodeling. Here a mechanism to directly supply lipids from the ER to the inner membrane complex (I...
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Reposted by Xinbo Wang
Michael Hanna @mike-hanna.bsky.social · 12/09/2025
New paper alert! I’m very excited to share our paper published in @embojournal.org! @pdc-lab.bsky.social @yaleneuro.bsky.social
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Reposted by Xinbo Wang
The Science of Parkinson's @scienceofpd.bsky.social · 10/04/2025
New research from @pdc-lab.bsky.social & collaborators finds that the bridge-like lipid transport protein VPS13C/PARK23 mediates ER–lysosome contacts following lysosome damage; #Parkinsons-associated LRRK2 is also recruited but at later stage www.nature.com/articles/s41...
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Reposted by Xinbo Wang
Nature Cell Biology @natcellbio.nature.com · 01/05/2025
☕Wang et al. show the recruitment of the #lipid channel protein VPS13C and formation of VPS13C-dependent contacts between endoplasmic reticulum and #lysosomes after lysosomal damage. bit.ly/4jT01lz
bit.ly
The bridge-like lipid transport protein VPS13C/PARK23 mediates ER–lysosome contacts following lysosome damage - Nature Cell Biology
Wang et al. show the recruitment of the lipid channel protein VPS13C and formation of VPS13C-dependent contacts between endoplasmic reticulum and lysosomes after lysosomal damage.
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Reposted by Xinbo Wang
De Camilli Lab @pdc-lab.bsky.social · 10/04/2025
"The bridge-like lipid transport protein VPS13C/PARK23 mediates ER–lysosome contacts following lysosome damage" out now in Nature Cell Biology rdcu.be/ehbaL
rdcu.be
The bridge-like lipid transport protein VPS13C/PARK23 mediates ER–lysosome contacts following lysosome damage
Nature Cell Biology - Wang et al. show the recruitment of the lipid channel protein VPS13C and formation of VPS13C-dependent contacts between endoplasmic reticulum and lysosomes after lysosomal...
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