iiinova — where your digital self keeps evolving

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Kelly Dawe @corncolors.bsky.social · 01/10/2026

The UGA Genetics Department is hiring! Please help spread the word. We have an opening for an Associate Professor/Professor in Genetics, Synthetic Biology, and Biological Engineering. See ad for details – and join us here in Athens, GA!

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Sarah Gilmour @sarahgilmour.bsky.social · 13/05/2026

🚨Preprint alert! If you you’ve been following the centromere field recently, you’ve seen some (excellent) work showing that repeat copy number is highly variable in centromere region leading to massive variation in size. Here we try to understand how that variation impacts centromere functionality:

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Katie Billmyre @katiebillmyre.bsky.social · 05/03/2026

Wanted to advertise the presentations from my lab occurring at #Dros26! It’s my first time getting to bring my trainees and can’t wait for them to share their work.

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Mary Goll @gollmg.bsky.social · 03/11/2025

UGA Genetics is hiring! Please share! An ideal candidate will employ genetic models of disease in a research program with an emphasis on epigenetics, aging, chromosome biology, gene regulation, systems biology, or drug resistance mechanisms. jobs.sciencecareers.org/job/675651/a...

jobs.sciencecareers.org

Assistant Professor in Genetics - Athens, Georgia (US) job with The University of Georgia Department of Genetics | 675651

Tenure-track Assistant Professor in Genetics

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The Rokas Lab at Vanderbilt University @rokaslab.bsky.social · 31/10/2025

Registrations and abstract submissions are now open for the always stellar #fungal #genetics conference at lovely Asilomar, CA - March 17-22, 2026. Thank you to @genetics-gsa.bsky.social for the amazing partnership with the fungal community. Hope to see you there!

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Casey terHorst @ecoevolab.bsky.social · 08/09/2025

The GRFP lives!! (due end of October) www.nsf.gov/funding/oppo...

nsf.gov

NSF Graduate Research Fellowship Program (GRFP)

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John M. Drake @jdrakephd.bsky.social · 21/08/2025

Given the tremendous diversity of microorganisms in the world, why do only a small fraction cause disease? My essay in #PLOSBiology argues that the answer is lack of opportunity and that humans encounter only one new bacterial pathogen for every 1.4b years lived. journals.plos.org/plosbiology/...

journals.plos.org

Why are there so few pathogens? Ecology and evolution in pathogen emergence

Why are there so few pathogens, and what determines their emergence? This Perspective argues that ecological and evolutionary forces (host availability, geographic exposure and microbial innovation) w...

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Jason Stajich @hyphaltip.bsky.social · 21/08/2025

Assistant/ Associate Professor of Mycology at UGA - Go be part of a Legendary Fungal Group fungi.franklinresearch.uga.edu and dept of plant pathology plantpath.caes.uga.edu www.ugajobsearch.com/postings/445...

ugajobsearch.com

Assistant or Associate Professor - Fungal Biology/Mycology

The Department of Plant Pathology in the College of Agricultural and Environmental Sciences (CAES) at the University of Georgia is recruiting for the position of Assistant or Associate Professor in Fu...

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Blake Billmyre @blakebillmyre.bsky.social · 21/08/2025

Come work with Fungal Group at UGA!

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PLOS Biology @plosbiology.org · 23/05/2025

#Fungal infections are hard to treat due to #DrugResistance. @blakebillmyre.bsky.social &co use a high-throughput #TNseq system in #Cryptococcus neoformans to identify >1400 essential genes & reveal a role for #mitochondrial genes in #fluconazole sensitivity @plosbiology.org 🧪 plos.io/4dz3iVm

Schematic of TN-seq in C. neoformans. Top left: Transposon insertions (orange arrow) into nonessential genes results in viable cells. In contrast, insertions into essential genes will result in dead and nonrecoverable cells. Top right: TN-seq works by generating a library of cells where each cell has a single independent transposon insertion in a random location. As in A, those insertions into essential regions cause the cells to die and are nonrecoverable. As a result, the total library (bottom) is depleted in insertions in essential regions. Bottom: The Ac/Ds transposon was split into an Ac transposase and a Ds transposon containing a neomycin resistance marker. This Ds transposon was integrated into an intron of URA5 and the Ac transposase was integrated into the safe haven locus. The resulting stain is ura− and neomycin resistant. Upon initiating transposition via growth on galactose, the strain becomes URA+ and mutant at another locus (depicted here as YFG1).

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Blake Billmyre @blakebillmyre.bsky.social · 13/10/2023

Enjoyed my first visit to Clemson for the CBEP/EPIC meeting. Great science and a really nice view!

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Rachel Helston @fungalmom.bsky.social · 02/10/2023

New paper on meiotic drive from the Zanders lab!

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