Antimicrobial tolerance and persistence are phenomena that enable pathogenic microbes to survive for extended periods in the presence of high concentrations of cidal drugs. Current evidence suggests t...

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Apply for Assistant/Associate/Full Professor, Non-Tenure Track, Infectious Diseases - Internal Medicine, McGovern Medical School job with UTHealth Houston in Texas, United States. Faculty & Physicians...

Antimicrobial tolerance and persistence are phenomena that enable pathogenic microbes to survive for extended periods in the presence of high concentrations of cidal drugs. Current evidence suggests t...

Imidazole propionate produced by gut microbiota is associated with atherosclerosis in mouse models and in humans, and causes the development of atherosclerosis through activation of the imidazoline-1 ...

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The term “reviewer fatigue” has become only too familiar in scientific publishing. This editorial discusses how we can ease the burden on reviewers to make the peer review system more sustainable, whi...

Mandimycin, a polyene macrolide, exhibits strong antifungal activity and possesses a mode of action that is distinct from other compounds of this class.

Imaging study reveals how fungal networks are constructed.

Antimicrobial tolerance and persistence are definitions that describe the ability of microorganisms to survive the action of antimicrobial drugs for extended periods. These phenomena are distinct from...

We are looking for an NSF-funded post-doc in our lab in the Department of Integrative Biology at the University of Texas at Austin. The work will examine immune mechanisms that potentially underlie…

Sulfur metabolism is an essential aspect of fungal physiology and pathogenicity. Fungal sulfur metabolism comprises anabolic and catabolic routes that are not well conserved in mammals, therefore i...

Sulfur metabolism is an essential aspect of fungal physiology and pathogenicity. Fungal sulfur metabolism comprises anabolic and catabolic routes that are not well conserved in mammals, therefore i...

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Collateral-sensitivity-based drug cycling effectively prevents and impedes the evolution of antifungal drug resistance in Candida auris.