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Franck Martin @eriani-martinlab.bsky.social · 14/09/2026

What does the ribosome need to initiate translation? - an AUG codon. Yes but not only ! - a Kozak context. In fact, it is much more complicated than that and different in every species ! Check out our latest publication: doi.org/10.1016/j.ce... @cnrs-alsace.bsky.social @cnrs.fr @unistra.fr

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Reposted by Franck Martin

Sciences et Avenir @sciencesetavenir.bsky.social · 18/02/2026

Dans cette terrible maladie, les motoneurones, cellules à l’origine des mouvements volontaires, se dégradent, entraînant une paralysie progressive. Ce sont des protéines qui sont en jeu. Et on sait maintenant précisément comment elles sont produites ! 👉 l.sciencesetavenir.fr/9O7

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Reposted by Franck Martin

Karim Majzoub @karimaj.bsky.social · 09/02/2026

Kudos to @eriani-martinlab.bsky.social and team for this important discovery !

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Reposted by Franck Martin

CNRS @cnrs.fr · 09/02/2026

#PressRelease🗞️ A specific molecular mechanism has been identified as the cause of the most frequent genetic forms of amyotrophic lateral sclerosis (ALS, also known as Charcot’s disease) and frontotemporal dementia (FTD). 👉 www.cnrs.fr/en/press/discov...

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Franck Martin @eriani-martinlab.bsky.social · 06/02/2026

I'm thrilled to anounce that our latest study on ALS/FTD neurodegenerative diseases has been published. Very proud to be part of this excting project. Many thanks to Clotilde Lagier Tourenne for an amazing longlasting and fruitfull collaboration, #ALS #FTD #Ribosomes www.science.org/doi/10.1126/...

science.org

Blocking RAN translation without altering repeat RNAs rescues C9ORF72-related ALS and FTD phenotypes

GGGGCC (G4C2) repeat expansion in C9ORF72 is the most common genetic cause of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). Toxicity is thought to result from the accumulation...

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Franck Martin @eriani-martinlab.bsky.social · 14/10/2025

Congratulations to Yann Tardivat for his PhD defense last Friday October 10th on the translation mechanisms during SARS-CoV-2 infection. A collaborative project with @novalix_pharma ! #sars @ARN67IBMC @unistra

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Franck Martin @eriani-martinlab.bsky.social · 14/10/2025

Last Wednesday October 8th, Antoine Perret defended his PhD entitled: Investigation of Tau mRNA translation mechanisms in Alzheimer’s Disease patients. Congratulations for this great achievement. A great collaborative project with @LucBuee and Malika Hamdane from Lille University

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Franck Martin @eriani-martinlab.bsky.social · 13/07/2025

Congratulations to Augustin Burn who tremendously succeeded in the grant competition for a PhD fellowship, welcome to the lab Augustin, we are looking forward to working with you!!!!

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Franck Martin @eriani-martinlab.bsky.social · 21/01/2025

I’ve just done my #eXit! the X exodus is massive. Don't lose any of your followers. Thanks to #HelloQuitX I've registered 338 new passengers for a journey to #BlueSky & #Mastodon. Join us on app.helloquitx.com and automatically find your communities on #January20!

app.helloquitx.com

HelloQuitteX

Libérez vos espaces numériques

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Reposted by Franck Martin

Luc Buée @lucbuee.bsky.social · 11/01/2025

Mapt mutations are found in brain disorders. Most of them are located in sequences encoding microtubule-binding domains. However, some are in the 5'UTR tau mRNAs. Understanding the effect of mutations in the amino-terminal part of tau and the role of N-truncated tau academic.oup.com/narmolmed/ar...

academic.oup.com

FTLD-MAPT mutations and short 5′UTR Tau mRNAs increase Tau translation

Abstract. Tau protein plays a central role in Alzheimer’s disease (AD) and other tauopathies. Tau proteins aggregate in degenerating neurons and their expr

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Reposted by Franck Martin

David Chavalarias @chavalarias.org · 07/01/2025

🔥🔥🔥 Notre app #HelloQuitteX est sortie ! Récupérez vos followers de X en moins de 5 min, il suffit de récupérer votre archive X. Le #20Janvier, on vous reconnecte avec tous vos abonnés de X ! Yes we can ! Rejoignez ➡️ app.helloquitx.com Dessin de @cartooningforpeace.bsky.social

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